Myositis ossificans
From Wikipedia, the free encyclopedia
 |
This article appears to contradict the article fibrodysplasia ossificans progressiva. Please see discussion on the linked talk page. |
 |
The factual accuracy of this article is disputed. Please see the relevant discussion on the talk page. (April 2008) |
 |
This article does not cite any references or sources. (April 2008) Please help improve this article by adding citations to reliable sources. Unverifiable material may be challenged and removed. |
| Myositis ossificans Classification and external resources |
|
| ICD-10 | M61. |
|---|---|
| ICD-9 | 728.1 |
| MeSH | D009221 |
Myositis ossificans comprises two syndromes characterized by heterotopic ossification (calcification) of muscle.
[edit] Classification
- In the first, and by far most common type, nonhereditary myositis ossificans (commonly referred to simply as "myositis ossificans", as in the remainder of this article), calcifications occur at the site of injured muscle, most commonly in the arms or in the quadriceps of the thighs.
- The second condition, myositis ossificans progressiva (also referred to as fibrodysplasia ossificans progressiva) is an inherited affliction, autosomal dominant pattern, in which the ossification can occur without injury, and typically grows in a predictable pattern.
[edit] Presentation
Myositis ossificans usually presents with pain, tenderness, focal swelling, and joint muscle reduction, in the aftermath of a painful muscle contusion which resolved more slowly than expected, if at all.
The condition rarely is asymptomatic, and may sometimes be diagnosed from radiographs obtained for unrelated problems.
Most (i.e. 80%) ossifications arise in the thigh or arm, and are caused by a premature return to activity after an injury. Other sites include intercostal spaces, erector spinae, pectoralis muscles, glutei, and the chest. Hazy densities are sometimes noted ca. one month after injury, while the denser opacities eventually seen may not be apparent until two months have passed
Treatment is initially conservative, as some patients' calcifications will spontaneously be reabsorbed, and others will have minimal symptoms. In occasional cases, surgical debridement of the abnormal tissue is required, although success of such therapy is limited.
